Still Free

Yeah, Mr. Smiley. Made it through the entire Trump presidency without being enslaved. Imagine that.

Monday, May 03, 2021

A Vascular Disease

As regular readers know, here at the Ghost we do facts and data. When we said Zinc is effective for Influenza and Wuhan, we posted the papers. When we said that Quercetin is effective, we posted the papers. When we told you that masks were ineffective, we showed you the physics. When we told you about the effectiveness of vitamin D, we posted the data. We have been wary of the recent "vaccines" because they have not been fully tested and some are possibly very dangerous because they tell the body to produce a protein that is actually deadly to the body. This post is about further evidence of this.

Now, if you recall there are a set of CV patients who have had fatigue as their major symptom. Often this fatigue is not accompanied by or resultant of any respiratory disfunction. I have been very curious as to why this is. Now we have an answer.

The paper, published on April 30, 2021, in Circulation Research, also shows conclusively that COVID-19 is a vascular disease, demonstrating exactly how the SARS-CoV-2 virus damages and attacks the vascular system on a cellular level. The findings help explain COVID-19’s wide variety of seemingly unconnected complications, and could open the door for new research into more effective therapies.


Note that this paper is asserting that Covid is a vascular disease rather than a respiratory disease. 

“That could explain why some people have strokes, and why some people have issues in other parts of the body. The commonality between them is that they all have vascular underpinnings.”

Lungs have a lot of small blood vessels. Kidneys have a lot of blood vessels.

In the new study, the researchers created a “pseudovirus” that was surrounded by SARS-CoV-2 classic crown of spike proteins, but did not contain any actual virus. Exposure to this pseudovirus resulted in damage to the lungs and arteries of an animal model—proving that the spike protein alone was enough to cause disease. Tissue samples showed inflammation in endothelial cells lining the pulmonary artery walls. [my underlines]

Special attention to the underlined portion. The spike protein itself can cause disease. Are bells ringing yet?

No? Well look here from the CDC:

Screenshot in case they change the text at a later date:



COVID-19 mRNA vaccines give instructions for our cells to make a harmless piece of what is called the “spike protein.” The spike protein is found on the surface of the virus that causes COVID-19.

"harmlesss what"? How exactly do they know it is "harmless"? Given the number of adverse reactions, "harmless" would be a relative term.

Anyway, back to the topic.

"The team then replicated this process in the lab, exposing healthy endothelial cells (which line arteries) to the spike protein. They showed that the spike protein damaged the cells by binding ACE2. This binding disrupted ACE2’s molecular signaling to mitochondria (organelles that generate energy for cells), causing the mitochondria to become damaged and fragmented."

For those who don't know, mitochondria are the "powerhouse" of the cell. If they go, well you're going to be very tired.

So now we can understand why there are people reporting fatigue sans other symptoms. Furthermore, what is "long-COVID" may in fact by remnants of these spike proteins continuing to do damage to mitochondrion.

Now lets go back to a point about vitamin D and the ACE2 receptor. From the NIH:

It has been known that angiotensin-converting enzyme 2 (ACE2) is the main host cell receptor of COVID-19. ACE2 is expressed in type II alveolar cells of the lungs, absorptive enterocytes from the ileum and colon, esophagus upper and stratified epithelial, kidney proximal tubule cells, myocardial cells, bladder urothelial cells, and epithelial cells of the oral mucosa (Zou et al. ; Xu et al. ). The high expression of ACE2 could be a potential risk factor for infection routes of COVID-19. 

And now:

Vitamin D has been known to play a critical role in the immune system. Vitamin D receptor has been expressed in multiple organs and tissues including the heart, lungs, kidneys, liver, nervous system, intestine, bone, parathyroid gland, cardiovascular system, and myocardium (Prufer et al. ).  


So they BEEN KNOWN that vitamin D plays critical immune roles in the very systems affected by Wuhan and yet they have NOT to this day, told you to take it or at a minimum to get your levels checked. And to add insult to injury got you to stay indoors for weeks and months so your body couldn't make its own. But now they have a vaccine to give you.

n human with primary and secondary kidney disease and in mice with diabetes, increased ACE2 expression was demonstrated (Ye et al. ; Lely et al. ). Vitamin D treatment was shown to inhibit ACE2 expression in the kidney (Ali et al. ). 1,25-(OH)2D3 exhibited renoprotective effect by decreasing ACE1 and ACE1/ACE2 ratio in streptozotocin-induced diabetic nephropatic rats (Lin et al. ). In other words, vitamin D treatment can suppress ACE2 expression in kidney tubule cells, thereby preventing COVID-19 entry into the cell in diabetic patients and protecting the kidney.


So for the Kidneys reducing ACE2 is beneficial. Known since 2018.

Vitamin D deficiency is an important risk factor in ARDS as well as in many diseases. A single high-dose preoperative treatment with oral vitamin D was reported to prevent ARDS by reducing postoperative pulmonary vascular permeability index (Parekh et al. ). A study showed that vitamin D exhibits direct protective effect on alveolar epithelium, and decreased the death of the type 2 alveolar epithelial cells in lipopolysaccharide-induced ARDS mouse model; also, supplementation of vitamin D before the oesophagectomy prevents ARDS by reducing alveolar capillary damage, in clinical setting (Dancer et al. ). Another study demonstrated that vitamin D treatment was shown to have protective effect on the lungs by inhibiting renin, ACE, and Ang II level, and increased ACE2 level expression in acute lung injury model in animal (Xu et al. ). Calcitriol (1,25-dihydroxyvitamin D3) may enhance the expression of ACE2 by pronouncedly impact on ACE2/Ang(1–7)/MasR pathway (Cui et al. ).

In the lungs, increased ACE2 is beneficial. Vitamin D is not a one trick pony.

So the question is why, given how much info has been known about vitamin D, Zinc and Quercetin, has this information not been heavily promoted to the public?  Why has any alternative to "vaccine" been viciously repressed?

These vaccines, which may be free to you as in immediate "out of pocket" but are not at all FREE, could not get emergency authorization IF there was an effective therapeutic available. If there was, the regular testing process, which could take years, would apply. There's no money in that. So the state and private industry has colluded, at a cost of many lives, to prevent alternative treatments so that two things could happen:

1) The companies get paid. 

2) The state could assume powers, particularly in western countries that they otherwise could never get via elections or legislative means.

Years ago I asked how many American citizens was Hillary Clinton willing to have killed for her "immigration policies". Let me expand that. Back in 2001 it was a conspiracy theory to say that certain members of the government, knew at least some of the 9-11 hijackers, and they "allowed" 9-11 to happen for their own ends. But the thought that US officials would allow nearly 3000 people to die for political or economic reasons, was too much for most. Now think over 500k Americans have died of (for the sake of argument we won't get into "with") COVID. Much of that could have been prevented by getting people on the items mentioned above and the widespread use of Ivermectin as  prophylaxis. It should no longer be an "out there" thought to consider that the government will kill US Citizens for money and power and a lot of useful idiots would go along with it.

https://www.salk.edu/news-release/the-novel-coronavirus-spike-protein-plays-additional-key-role-in-illness/